Product Description Research Compound TUDCA 500 mg Capsules Research-grade capsule formulation supplied in a sealed bottle. TUDCA (tauroursodeoxycholic acid) is a conjugated hydrophilic bile acid studied in controlled laboratory settings for its relationship to ER stress modulation, hepatocellular protection, mitochondrial stability, apoptotic signaling, and bile-acid-associated cellular homeostasis. Research Use Only This product is intended strictly for laboratory and scientific investigation. Not for human consumption, therapeutic use, or veterinary application. Specifications Form Capsules Content 500 mg TUDCA per capsule Quantity 60 capsules Packaging Sealed bottle Storage Store at room temperature, protected from light, keep desiccated Molecular Formula C26H45NO6S Molecular Weight ~515.7 g·mol⁻¹ IUPAC Name 2-[[(3α,5β,7β)-3,7-dihydroxy-24-oxocholan-24-yl]amino]ethanesulfonic acid CAS Number 14605-22-2 Research Overview TUDCA is studied as a bile-acid-derived research compound with strong interest in ER stress modulation, mitochondrial stability, and hepatocellular support pathways. In experimental settings, it is investigated for its relationship to unfolded protein response signaling, membrane stability, apoptosis-related mechanisms, cholestatic models, and broader cell-survival pathways. No therapeutic or clinical claims are made or implied. Primary Research Areas ER Stress & UPR Signaling Experimental investigation of proteostasis, misfolded-protein burden, and unfolded protein response pathways including stress-adaptation signaling. Hepatocellular Protection Preclinical research focused on liver-cell integrity, bile-acid homeostasis, cholestatic processes, and hepatobiliary stress models. Mitochondrial Function & Apoptosis Laboratory studies examining membrane potential stability, cytochrome c release, apoptotic signaling, and cellular resilience under stress conditions. Neuroprotection & Cell Survival Models Research involving neuronal survival, stress-response signaling, and broader cytoprotective laboratory pathways beyond liver-focused models.
